DBQD2_XYLT1

Gene
XYLT1
Disease
DBQD2
Inheritance
AR
Classification
Moderate
Total Score
8.5
Publications Reviewed
2
Publication Span
4.83 years
Last Updated
08/18/2025
Curator(s)
Macayla Weiner, Laurel Hiatt

Description

Autosomal recessive DBQD2/Baratela-Scott syndrome is associated with biallelic XYLT1 pathogenic variants, including a promoter/5' UTR GGC repeat expansion that causes XYLT1 exon 1 hypermethylation and transcriptional silencing. XYLT1 encodes xylosyltransferase I, which initiates proteoglycan glycosaminoglycan-chain biosynthesis; gene-level patient fibroblast studies show reduced XYLT1 expression and impaired cellular proteoglycan synthesis, supporting disease relevance but not always repeat-specific mechanism.

Genetic evidence

Total: 6

Singular EvidenceProbandsPMID:305547216Twelve affected individuals from 10 unrelated BSS/DBQD2-spectrum families were investigated; XYLT1 pathogenic alleles included biallelic sequence/CNV variants and a promoter/5' UTR GGC repeat expansion associated with exon 1 hypermethylation. The methylated/expanded allele accounted for 50% of disease alleles and segregated in trans with sequence variants or deletions where assessable.
1 rows

Experimental evidence

Total: 2.5

FunctionBiochemical functionPMID:245817410.5Gene-level, not tandem-repeat-specific: XYLT1 encodes XT-I, which catalyzes the first step of proteoglycan biosynthesis by transferring xylose to serine residues of proteoglycan core proteins to initiate GAG-chain synthesis.
FunctionProtein interactionPMID:245817410.5PMID 24581741 describes XT-I enzymatic function and effects on proteoglycan biosynthesis.
FunctionRegulatory impactPMID:245817411.5Gene-level, not tandem-repeat-specific: qPCR in two patient fibroblast lines with truncating XYLT1 variants showed a dramatic reduction of XYLT1 cDNA; XYLT2 expression was decreased compared with one control, whereas B4GALT7 was not affected.
3 rows

Note: Maximum score caps apply at evidence type, category, and supercategory levels, so section totals may be lower than the raw sum of row scores.